Deficit of p66ShcA Restores Redox-sensitive Stress Response Program in Cisplatin-induced Acute Kidney Injury

Overwhelming oxidative stress and compromised tubular cell antioxidant response have been incriminated for cisplatin (Cis) -induced acute kidney injury (AKI). We hypothesized that Cis-induced KI was the outcome of the deactivated redox-sensitive stress response program (RSSRP). Wild (WT) and heteroz...

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Detalhes bibliográficos
Main Authors: Rattanavich, Rungwasee, Plagov, Andrei, Kumar, Dileep, Rai, Partab, Lederman, Rivka, Salhan, Divya, Vashistha, Himanshu, Malhotra, Ashwani, Meggs, Leonard G., Singhal, Pravin C
Formato: Artigo
Idioma:Inglês
Publicado em: 2013
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3720132/
https://ncbi.nlm.nih.gov/pubmed/23506954
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yexmp.2013.03.001
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