Persistent Reversal of Enhanced Amphetamine Intake by Transient CaMKII Inhibition

Amphetamine exposure transiently increases Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) α expression in the nucleus accumbens (NAcc) shell and this persistently increases local GluA1 S831 phosphorylation and enhances behavioral responding to the drug. Here we assessed whether transiently i...

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Hlavní autoři: Loweth, Jessica A., Li, Dongdong, Cortright, James J., Wilke, Georgia, Jeyifous, Okunola, Neve, Rachael L., Bayer, K. Ulrich, Vezina, Paul
Médium: Artigo
Jazyk:Inglês
Vydáno: Society for Neuroscience 2013
Témata:
Brief Communications
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3710147/
https://ncbi.nlm.nih.gov/pubmed/23345217
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4386-13.2013
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