SAD-A Potentiates Glucose-Stimulated Insulin Secretion as a Mediator of Glucagon-Like Peptide 1 Response in Pancreatic β Cells

समान संसाधन

• Synapses of Amphids Defective (SAD-A) Kinase Promotes Glucose-stimulated Insulin Secretion through Activation of p21-activated Kinase (PAK1) in Pancreatic β-Cells
  द्वारा: Nie, Jia, और अन्य
  प्रकाशित: (2012)
• SAD kinases sculpt axonal arbors of sensory neurons through long and short-term responses to neurotrophin signals
  द्वारा: Lilley, Brendan N., और अन्य
  प्रकाशित: (2013)
• SAD-A kinase controls islet β-cell size and function as a mediator of mTORC1 signaling
  द्वारा: Nie, Jia, और अन्य
  प्रकाशित: (2013)
• SAD-A Promotes Glucose-Stimulated Insulin Secretion Through Phosphorylation and Inhibition of GDIα in Male Islet β Cells
  द्वारा: Nie, Jia, और अन्य
  प्रकाशित: (2018)
• Somatostatin and insulin mediate glucose-inhibited glucagon secretion in the pancreatic α-cell by lowering cAMP
  द्वारा: Elliott, Amicia D., और अन्य
  प्रकाशित: (2014)