Murine cell complementation of a cold-sensitive defect for simian virus 40 replication in simian cells.

Mouse 3T3 fibroblasts were found to complement, in simian cell variants semipermissive to simian virus 40, a cold-sensitive defect of an early function, but not a nonconditional defect of viral uncoating. The variant simian cells could rescue simian virus 40 from 3T3 transformants, and this capacity...

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Vydáno v:	Mol Cell Biol
Hlavní autoři:	Fischer-Fantuzzi, L, Vesco, C
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	Taylor & Francis 1982
Témata:	Research Article
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC369909/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/6294506/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.2.9.1155-1157.1982
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