SMAD3 Deficiency Promotes Inflammatory Aortic Aneurysms in Angiotensin II–Infused Mice Via Activation of iNOS

BACKGROUND: Ninety percent of the patients carrying distinct SMAD3 mutations develop aortic aneurysms and dissections, called aneurysms‐osteoarthritis syndrome (AOS). However, the etiology and molecular events downstream of SMAD3 leading to the pathogenesis of aortic aneurysms in these patients stil...

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Detaylı Bibliyografya
Asıl Yazarlar: Tan, Chek K., Tan, Eddie H., Luo, Baiwen, Huang, Charlotte L., Loo, Joachim S., Choong, Cleo, Tan, Nguan S.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Blackwell Publishing Ltd 2013
Konular:
Original Research
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3698794/
https://ncbi.nlm.nih.gov/pubmed/23782924
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.113.000269
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