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The myeloid response to pancreatic carcinogenesis is regulated by the receptor for advanced glycation end-products
We identified a critical role for receptor for advanced glycation end products (RAGE) in the intratumoral accumulation of myeloid-derived suppressor cells (MDSCs) during pancreatic carcinogenesis. The absence of RAGE markedly delayed neoplasia and limited MDSC accumulation in mice expressing an onco...
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| Main Authors: | , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Landes Bioscience
2013
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3667906/ https://ncbi.nlm.nih.gov/pubmed/23762800 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/onci.24184 |
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