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Deregulated G1–S control and energy stress contribute to the synthetic-lethal interactions between inactivation of RB and TSC1 or TSC2

Synthetic lethality is a potential strategy for cancer treatment by specifically promoting the death of cancer cells with particular defects such as the loss of the RB (RB1) tumor suppressor. We previously showed that inactivation of both RB and TSC2 induces synergistic apoptosis during the developm...

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Detalles Bibliográficos
Main Authors: Gordon, Gabriel M., Zhang, Tianyi, Zhao, Jiong, Du, Wei
Formato: Artigo
Idioma:Inglês
Publicado: The Company of Biologists 2013
Assuntos:
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC3666254/
https://ncbi.nlm.nih.gov/pubmed/23447678
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/jcs.121301
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