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Notch signaling maintains Leydig progenitor cells in the mouse testis
During testis development, fetal Leydig cells increase their population from a pool of progenitor cells rather than from proliferation of a differentiated cell population. However, the mechanism that regulates Leydig stem cell self-renewal and differentiation is unknown. Here, we show that blocking...
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Main Authors: | , , , , , |
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Formato: | Artigo |
Idioma: | Inglês |
Publicado em: |
2008
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Assuntos: | |
Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3653410/ https://ncbi.nlm.nih.gov/pubmed/18927153 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/dev.024786 |
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