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Loss of the K(v)1.1 potassium channel promotes pathologic sharp waves and high frequency oscillations in in vitro hippocampal slices
In human disease, channelopathies involving functional reduction of the delayed rectifier potassium channel α-subunit K(v)1.1 – either by mutation or autoimmune inhibition – result in temporal lobe epilepsy. K(v)1.1 is prominently expressed in the axons of the hippocampal tri-synaptic pathway, sugge...
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| Main Authors: | , , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2013
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3648846/ https://ncbi.nlm.nih.gov/pubmed/23466697 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2013.02.009 |
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