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C5a is not involved in experimental autoimmune myasthenia gravis pathogenesis
C5 deficient mice are highly resistant to experimental autoimmune myasthenia gravis (EAMG) despite intact immune response to acethylcholine receptor (AChR), validating the pivotal role played by membrane attack complex (MAC, C5b-9) in neuromuscular junction destruction. To distinguish the significan...
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| Autori principali: | , , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2008
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3645869/ https://ncbi.nlm.nih.gov/pubmed/18455242 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jneuroim.2008.03.007 |
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