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IL-33–dependent induction of allergic lung inflammation by FcγRIII signaling
Atopic asthma is a chronic inflammatory disease of the lungs generally marked by excessive Th2 inflammation. The role of allergen-specific IgG in asthma is still controversial; however, a receptor of IgG–immune complexes (IgG-ICs), FcγRIII, has been shown to promote Th2 responses through an unknown...
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| Main Authors: | , , , , , , , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
American Society for Clinical Investigation
2013
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3635716/ https://ncbi.nlm.nih.gov/pubmed/23585480 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI63802 |
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