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RAD53 is limiting in double-strand break repair and in protection against toxicity associated with ribonucleotide reductase inhibition
The yeast Chk2/Chk1 homolog Rad53 is a central component of the DNA damage checkpoint system. While it controls genotoxic stress responses such as cell cycle arrest, replication fork stabilization and increase in dNTP pools, little is known about the consequences of reduced Rad53 levels on the vario...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3631316/ https://ncbi.nlm.nih.gov/pubmed/22277748 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.dnarep.2011.12.008 |
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