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Somatostatin type-2 receptor activation inhibits glutamate release and prevents status epilepticus
Newer therapies are needed for the treatment of status epilepticus (SE) refractory to benzodiazepines. Enhanced glutamatergic neurotransmission leads to SE, and AMPA receptors are modified during SE. Reducing glutamate release during SE is a potential approach to terminate SE. The neuropeptide somat...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2013
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3628955/ https://ncbi.nlm.nih.gov/pubmed/23473742 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2013.02.015 |
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