Mice Devoid of Tau Have Increased Susceptibility to Neuronal Damage in Myelin Oligodendrocyte Glycoprotein-Induced Experimental Autoimmune Encephalomyelitis

Lignende værker

• Loss of the receptor tyrosine kinase Axl leads to enhanced inflammation in the CNS and delayed removal of myelin debris during Experimental Autoimmune Encephalomyelitis
  af: Weinger, Jason G, et al.
  Udgivet: (2011)
• Suppression of inflammatory responses during MOG-induced experimental autoimmune encephalomyelitis is regulated by AKT3 signaling
  af: Tsiperson, Vladislav, et al.
  Udgivet: (2013)
• Damage to the Optic Chiasm in Myelin Oligodendrocyte Glycoprotein–Experimental Autoimmune Encephalomyelitis Mice
  af: Herrera, Sheryl L, et al.
  Udgivet: (2014)
• Polyreactive Myelin Oligodendrocyte Glycoprotein Antibodies: Implications for Systemic Autoimmunity in Progressive Experimental Autoimmune Encephalomyelitis
  af: Peterson, Lisa K., et al.
  Udgivet: (2007)
• Memory cells specific for myelin oligodendrocyte glycoprotein (MOG) transfer experimental autoimmune encephalomyelitis()
  af: Williams, Jessica L, et al.
  Udgivet: (2011)