In Vivo Suppression of MiR-24 Prevents the Transition toward Decompensated Hypertrophy in Aortic-constricted Mice

RATIONALE: During the transition from compensated hypertrophy to heart failure, the signaling between L-type Ca(2+) channels (LCCs) in the cell membrane/T-tubules (TTs) and ryanodine receptors (RyRs) in the sarcoplasmic reticulum (SR) becomes defective, partially due to the decreased expression of a...

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Bibliografiset tiedot
Päätekijät: Li, Rong-Chang, Tao, Jin, Guo, Yun-Bo, Wu, Hao-Di, Liu, Rui-Feng, Bai, Yan, Lv, Zhi-Zhen, Luo, Guan-Zheng, Li, Lin-Lin, Wang, Meng, Yang, Hua-Qian, Gao, Wei, Han, Qi-De, Zhang, You-Yi, Wang, Xiu-Jie, Xu, Ming, Wang, Shi-Qiang
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2013
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Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3622206/
https://ncbi.nlm.nih.gov/pubmed/23307820
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.112.300806
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