Pharmacological inhibition of S-nitrosoglutathione reductase improves endothelial vasodilatory function in rats in vivo

Nitric oxide (NO) exerts a wide range of cellular effects in the cardiovascular system. NO is short lived, but S-nitrosoglutathione (GSNO) functions as a stable intracellular bioavailable NO pool. Accordingly, increased levels can facilitate NO-mediated processes, and conversely, catabolism of GSNO...

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Xehetasun bibliografikoak
Egile Nagusiak: Chen, Qiumei, Sievers, Richard E., Varga, Monika, Kharait, Sourabh, Haddad, Daniel J., Patton, Aaron K., Delany, Christopher S., Mutka, Sarah C., Blonder, Joan P., Dubé, Gregory P., Rosenthal, Gary J., Springer, Matthew L.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Physiological Society 2013
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Articles
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC3615603/
https://ncbi.nlm.nih.gov/pubmed/23349456
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.01302.2012
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