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Complement Receptor 3 Influences Toll-like Receptor 7/8-Dependent Inflammation: IMPLICATIONS FOR AUTOIMMUNE DISEASES CHARACTERIZED BY ANTIBODY REACTIVITY TO RIBONUCLEOPROTEINS
Toll-like receptor (TLR) signaling is an important component in the inflammatory response generated in diseases characterized by autoantibody reactivity to proteins such as SSA/Ro in complex with endogenous nucleic acids. Complement receptor 3 (CR3), a genetic variant of which has been identified as...
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| 主要な著者: | , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Biochemistry and Molecular Biology
2013
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3610980/ https://ncbi.nlm.nih.gov/pubmed/23386618 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M112.403303 |
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