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IRF7-dependent IFNβ production in response to RANKL promotes medullary thymic epithelial cell development

The contributions of IRF3/7 and the type I interferons IFNα/β to the innate host defense have been extensively investigated, however, their role in thymic development is less clear. Here we show that mice lacking the type I interferon receptor IFNAR or the downstream transcription factor STAT1 harbo...

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Detalhes bibliográficos
Main Authors: Otero, Dennis C., Baker, Darren P., David, Michael
Formato: Artigo
Idioma:Inglês
Publicado em: 2013
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3608802/
https://ncbi.nlm.nih.gov/pubmed/23440417
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1203086
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