Leptin is key to peroxynitrite-mediated oxidative stress and Kupffer cell activation in experimental nonalcoholic steatohepatitis

BACKGROUND AND AIMS: Progression from steatosis to steatohepatitic lesions is hypothesized to require a second hit. These lesions have been associated with increased oxidative stress, often ascribed to high levels of leptin and other proinflammatory mediators. Here we have examined the role of lepti...

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Main Authors: Chatterjee, Saurabh, Ganini, Douglas, Tokar, Erik J., Kumar, Ashutosh, Das, Suvarthi, Corbett, Jean, Kadiiska, Maria, Waalkes, Michael, Diehl, Anna Mae, Mason, Ronald P.
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3596459/
https://ncbi.nlm.nih.gov/pubmed/23207144
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jhep.2012.11.035
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