Nonimmune thyroid destruction results from transgenic overexpression of an allogeneic major histocompatibility complex class I protein.
The overexpression of major histocompatibility complex (MHC) class I molecules in endocrine epithelial cells is an early feature of autoimmune thyroid disease and insulin-dependent diabetes mellitus, which may reflect a cellular response, e.g., to viruses or toxins. Evidence from a transgenic model...
Αποθηκεύτηκε σε:
| Εκδόθηκε σε: | Mol Cell Biol |
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| Κύριοι συγγραφείς: | , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Taylor & Francis
1993
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC359467/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8441397/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.13.3.1554-1564.1993 |
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