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Signaling via MYD88 in the pancreatic tumor microenvironment: A double-edged sword
We have recently shown that Toll-like receptor (TLR) signaling exacerbates pancreatic fibro-inflammation and promotes carcinogenesis in mice. Paradoxically, inhibition of the TLR-MYD88 signaling pathway is pro-tumorigenic owing to the dendritic cell-mediated T(H)2-polarization of CD4(+) T cells. TLR...
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| 主要な著者: | , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Landes Bioscience
2013
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3583926/ https://ncbi.nlm.nih.gov/pubmed/23483804 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/onci.22567 |
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