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Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells
BACKGROUND: Neoplastic transformation of cultured cells by a number of oncogenes such as src suppresses gap junctional, intercellular communication (GJIC); however, the role of Src and its effector Signal transducer and activator of transcription-3 (Stat3) upon GJIC in non small cell lung cancer (NS...
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| Autori principali: | , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
BioMed Central
2012
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3575370/ https://ncbi.nlm.nih.gov/pubmed/23244248 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2407-12-605 |
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