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Oxidative stress, fibrosis, and early afterdepolarization-mediated cardiac arrhythmias
Animal and clinical studies have demonstrated that oxidative stress, a common pathophysiological factor in cardiac disease, reduces repolarization reserve by enhancing the L-type calcium current, the late Na, and the Na-Ca exchanger, promoting early afterdepolarizations (EADs) that can initiate vent...
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| Hoofdauteurs: | , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Frontiers Media S.A.
2013
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3573324/ https://ncbi.nlm.nih.gov/pubmed/23423152 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphys.2013.00019 |
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