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Production of β-globin and adult hemoglobin following G418 treatment of erythroid precursor cells from homozygous β(0)39 thalassemia patients
In several types of thalassemia (including β(0)39-thalassemia), stop codon mutations lead to premature translation termination and to mRNA destabilization through nonsense-mediated decay. Drugs (for instance aminoglycosides) can be designed to suppress premature termination, inducing a ribosomal rea...
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| 主要な著者: | , , , , , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2009
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3572903/ https://ncbi.nlm.nih.gov/pubmed/19810011 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/ajh.21539 |
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