An immunoreceptor tyrosine-based inhibition motif in varicella-zoster virus glycoprotein B regulates cell fusion and skin pathogenesis

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• Disruption of PML nuclear bodies is mediated by ORF61 SUMO-interacting motifs and required for varicella-zoster virus pathogenesis in skin.
  gan: Li Wang, et al.
  Cyhoeddwyd: (2011-08-01)
• Disruption of PML Nuclear Bodies Is Mediated by ORF61 SUMO-Interacting Motifs and Required for Varicella-Zoster Virus Pathogenesis in Skin
  gan: Wang, Li, et al.
  Cyhoeddwyd: (2011)
• The C-terminus of varicella-zoster virus glycoprotein M contains trafficking motifs that mediate skin virulence in the SCID-human model of VZV pathogenesis
  gan: Leigh, Zerboni, et al.
  Cyhoeddwyd: (2018)
• Mutagenesis of Varicella-Zoster Virus Glycoprotein B: Putative Fusion Loop Residues Are Essential for Viral Replication, and the Furin Cleavage Motif Contributes to Pathogenesis in Skin Tissue In Vivo
  gan: Oliver, Stefan L., et al.
  Cyhoeddwyd: (2009)
• Structure–function analysis of varicella-zoster virus glycoprotein H identifies domain-specific roles for fusion and skin tropism
  gan: Vleck, Susan E., et al.
  Cyhoeddwyd: (2011)