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Arsenic Activates Endothelin-1 Gi Protein–Coupled Receptor Signaling to Inhibit Stem Cell Differentiation in Adipogenesis
Dysfunctional lipid and glucose metabolism contribute to metabolic syndrome—a major public health concern that enhances cardiovascular disease risk. Arsenic (As(III)) exposure may increase metabolic syndrome and cardiovascular disease risk by impairing adipose tissue differentiation, function, and i...
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| Autori principali: | , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Oxford University Press
2013
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3551431/ https://ncbi.nlm.nih.gov/pubmed/23152186 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/toxsci/kfs323 |
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