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Pathophysiology and Clinical Manifestations of the β-Thalassemias
The β-thalassemia syndromes reflect deficient or absent β-globin synthesis usually owing to a mutation in the β-globin locus. The relative excess of α-globin results in the formation of insoluble aggregates leading to ineffective erythropoiesis and shortened red cell survival. A relatively high capa...
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| Hoofdauteurs: | , |
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| Formaat: | Artigo |
| Taal: | Inglês |
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Cold Spring Harbor Laboratory Press
2012
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| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3543079/ https://ncbi.nlm.nih.gov/pubmed/23209183 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/cshperspect.a011726 |
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