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Normal Adaptation of Candida albicans to the Murine Gastrointestinal Tract Requires Efg1p-Dependent Regulation of Metabolic and Host Defense Genes

Although gastrointestinal colonization by the opportunistic fungal pathogen Candida albicans is generally benign, severe systemic infections are thought to arise due to escape of commensal C. albicans from the gastrointestinal (GI) tract. The C. albicans transcription factor Efg1p is a major regulat...

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Bibliografiset tiedot
Päätekijät: Pierce, Jessica V., Dignard, Daniel, Whiteway, Malcolm, Kumamoto, Carol A.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Microbiology 2013
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3535844/
https://ncbi.nlm.nih.gov/pubmed/23125349
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/EC.00236-12
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