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Normal Adaptation of Candida albicans to the Murine Gastrointestinal Tract Requires Efg1p-Dependent Regulation of Metabolic and Host Defense Genes

Although gastrointestinal colonization by the opportunistic fungal pathogen Candida albicans is generally benign, severe systemic infections are thought to arise due to escape of commensal C. albicans from the gastrointestinal (GI) tract. The C. albicans transcription factor Efg1p is a major regulat...

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Autori principali: Pierce, Jessica V., Dignard, Daniel, Whiteway, Malcolm, Kumamoto, Carol A.
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Microbiology 2013
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3535844/
https://ncbi.nlm.nih.gov/pubmed/23125349
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/EC.00236-12
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