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The regulation and role of neuronal gap junctions during neuronal injury
In the mammalian CNS, excessive release of glutamate and overactivation of glutamate receptors are responsible for the secondary (delayed) neuronal death following neuronal injury, including ischemia, traumatic brain injury (TBI) and epilepsy. The coupling of neurons by gap junctions (electrical syn...
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| Autor principal: | |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Landes Bioscience
2012
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3508779/ https://ncbi.nlm.nih.gov/pubmed/22976616 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/chan.21685 |
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