Neuronal Gap Junction Coupling Is Regulated by Glutamate and Plays Critical Role in Cell Death during Neuronal Injury

In the mammalian CNS, excessive release of glutamate and overactivation of glutamate receptors are responsible for the secondary (delayed) neuronal death following neuronal injury, including ischemia, traumatic brain injury (TBI), and epilepsy. The coupling of neurons by gap junctions (electrical sy...

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Detalhes bibliográficos
Main Authors: Wang, Yongfu, Song, Ji-Hoon, Denisova, Janna V., Park, Won-Mee, Fontes, Joseph D., Belousov, Andrei B.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2012
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3567463/
https://ncbi.nlm.nih.gov/pubmed/22238107
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3872-11.2012
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