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Acute hemodynamic effects of erythropoietin do not mediate its cardioprotective properties
Activation of nitric oxide (NO) signaling is considered, at list partially, a mechanistic basis for EPO-induced cardioprotection. Surprisingly, hemodynamic response subsequent to NO activation after EPO administration has never been reported. The objectives of this study were to evaluate the acute h...
Tallennettuna:
| Päätekijät: | , , |
|---|---|
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
The Company of Biologists
2012
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3507179/ https://ncbi.nlm.nih.gov/pubmed/23213383 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/bio.20122378 |
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