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BH3-only protein silencing contributes to acquired resistance to PLX4720 in human melanoma

B-RAF is mutated to a constitutively active form in 8% of human cancers including 50% of melanomas. In clinical trials, the RAF inhibitor, PLX4032 (vemurafenib), caused partial or complete responses in 48–81% of mutant B-RAF harboring melanoma patients. However, the average duration of response was...

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Detalhes bibliográficos
Main Authors: Shao, Y, Aplin, A E
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group 2012
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3504716/
https://ncbi.nlm.nih.gov/pubmed/22858545
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cdd.2012.94
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