Lack of CAK complex accumulation at DNA damage sites in XP-B and XP-B/CS fibroblasts reveals differential regulation of CAK anchoring to core TFIIH by XPB and XPD helicases during nucleotide excision repair

Lignende værker

• Dissociation of CAK from Core TFIIH Reveals a Functional Link between XP-G/CS and the TFIIH Disassembly State
  af: Arab, Hany H., et al.
  Udgivet: (2010)
• XPB and XPD helicases in TFIIH orchestrate DNA duplex opening and damage verification to coordinate repair with transcription and cell cycle via CAK kinase
  af: Fuss, Jill O., et al.
  Udgivet: (2011)
• Phosphorylation of XPB helicase regulates TFIIH nucleotide excision repair activity
  af: Coin, Frédéric, et al.
  Udgivet: (2004)
• Spironolactone-induced XPB degradation requires TFIIH integrity and ubiquitin-selective segregase VCP/p97
  af: Chauhan, Anil K., et al.
  Udgivet: (2020)
• Mutations in XPB and XPD helicases found in xeroderma pigmentosum patients impair the transcription function of TFIIH.
  af: Coin, F, et al.
  Udgivet: (1999)