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Particulate matter air pollution disrupts endothelial cell barrier via calpain-mediated tight junction protein degradation
BACKGROUND: Exposure to particulate matter (PM) is a significant risk factor for increased cardiopulmonary morbidity and mortality. The mechanism of PM-mediated pathophysiology remains unknown. However, PM is proinflammatory to the endothelium and increases vascular permeability in vitro and in vivo...
Tallennettuna:
Päätekijät: | , , , , , , , , , , , |
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Aineistotyyppi: | Artigo |
Kieli: | Inglês |
Julkaistu: |
BioMed Central
2012
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Aiheet: | |
Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3489700/ https://ncbi.nlm.nih.gov/pubmed/22931549 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1743-8977-9-35 |
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