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Human mitochondrial oxidative capacity is acutely impaired following burn trauma

BACKGROUND: Mitochondrial proteins and genes are damaged after burn injury in animals but have not previously been assessed in human burn patients. METHODS: The rates of maximal muscle mitochondrial oxidative capacity(ATP production) and uncoupled oxidation(heat production) for both palmitate and py...

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Detaylı Bibliyografya
Asıl Yazarlar: Cree, Melanie G., Fram, Ricki Y., Herndon, David N., Qian, Ting, Angel, Carlos, Green, Justin M., Mlcak, Ronald, Aarsland, Asle, Wolfe, Robert R.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2008
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3480314/
https://ncbi.nlm.nih.gov/pubmed/18639661
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.amjsurg.2007.09.048
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