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Human mitochondrial oxidative capacity is acutely impaired following burn trauma
BACKGROUND: Mitochondrial proteins and genes are damaged after burn injury in animals but have not previously been assessed in human burn patients. METHODS: The rates of maximal muscle mitochondrial oxidative capacity(ATP production) and uncoupled oxidation(heat production) for both palmitate and py...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2008
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3480314/ https://ncbi.nlm.nih.gov/pubmed/18639661 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.amjsurg.2007.09.048 |
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