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Modulation of K(2P)3.1 (TASK-1), K(2P)9.1 (TASK-3) and TASK-1/3 heteromer by reactive oxygen species
Reactive oxygen species (ROS) generated by mitochondria or NADPH oxidase have been implicated in the inhibition of K(+) current by hypoxia in chemoreceptor cells. As TASKs are highly active background K(+) channels in these cells, we studied the role of ROS in hypoxia-induced inhibition of TASKs. In...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2012
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3478902/ https://ncbi.nlm.nih.gov/pubmed/23007462 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00424-012-1159-y |
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