Prevention of Hepatocellular Adenoma and Correction of Metabolic Abnormalities in Murine Glycogen Storage Disease Type Ia by Gene Therapy

Samankaltaisia teoksia

• Minimal hepatic glucose-6-phosphatase-α activity required to sustain survival and prevent hepatocellular adenoma formation in murine glycogen storage disease type Ia
  Tekijä: Lee, Young Mok, et al.
  Julkaistu: (2015)
• The Upstream enhancer elements of the G6PC promoter are critical for optimal G6PC expression in murine glycogen storage disease type Ia
  Tekijä: Lee, Young Mok, et al.
  Julkaistu: (2013)
• Complete Normalization of Hepatic G6PC Deficiency in Murine Glycogen Storage Disease Type Ia Using Gene Therapy
  Tekijä: Yiu, Wai Han, et al.
  Julkaistu: (2010)
• Normoglycemia alone is insufficient to prevent long term complications of hepatocellular adenoma in glycogen storage disease type Ib mice
  Tekijä: Yiu, Wai Han, et al.
  Julkaistu: (2009)
• Activation of tumor-promoting pathways implicated in hepatocellular adenoma/carcinoma, a long-term complication of glycogen storage disease type Ia
  Tekijä: Cho, Jun-Ho, et al.
  Julkaistu: (2019)