ACE inhibition prevents diastolic Ca2+ overload and loss of myofilament Ca2+ sensitivity after myocardial infarction

Prevention of adverse cardiac remodeling after myocardial infarction (MI) remains a therapeutic challenge. Angiotensin-converting enzyme inhibitors (ACE-I) are a well-established first-line treatment. ACE-I delay fibrosis, but little is known about their molecular effects on cardiomyocytes. We inves...

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Hauptverfasser: Zalvidea, Santiago, Andre, Lucas, Loyer, Xavier, Cassan, Cécile, Sainte-Marie, Yannis, Thireau, Jérôme, Sjaastad, Ivar, Heymes, Christophe, Pasquié, Jean-Luc, Cazorla, Olivier, Aimond, Franck, Richard, Sylvain
Format: Artigo
Sprache:Inglês
Veröffentlicht: Bentham Science Publishers 2012
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Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3472404/
https://ncbi.nlm.nih.gov/pubmed/22280358
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