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Pendrin protein abundance in the kidney is regulated by nitric oxide and cAMP

Pendrin is a Cl(−)/HCO(3)(−) exchanger, expressed in the apical regions of some intercalated cell subtypes, and is critical in the pressor response to angiotensin II. Since angiotensin type 1 receptor inhibitors reduce renal pendrin protein abundance in mice in vivo through a mechanism that is depen...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Thumova, Monika, Pech, Vladimir, Froehlich, Otto, Agazatian, Diana, Wang, Xiaonan, Verlander, Jill W., Kim, Young Hee, Wall, Susan M.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Physiological Society 2012
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3468526/
https://ncbi.nlm.nih.gov/pubmed/22811483
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00577.2011
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