Adaptive resynthesis of O(6)-methylguanine-accepting protein can explain the differences between mammalian cells proficient and deficient in methyl excision repair

Lignende værker

• Adaptive increase of O6-methylguanine-acceptor protein in HeLa cells following N-methyl-N'-nitro-N-nitrosoguanidine treatment.
  af: Waldstein, E A, et al.
  Udgivet: (1982)
• Extracts of chronic lymphocytic leukemia lymphocytes have a high level of DNA repair activity fo O6-methylguanine.
  af: Waldstein, E A, et al.
  Udgivet: (1982)
• Role of ATP in Excision Repair of Ultraviolet Radiation Damage in Escherichia coli
  af: Waldstein, E. A., et al.
  Udgivet: (1974)
• Isolation of human complexes proficient in nucleotide excision repair.
  af: He, Z, et al.
  Udgivet: (1997)
• Mutations associated with base excision repair deficiency and methylation-induced genotoxic stress
  af: Sobol, Robert W., et al.
  Udgivet: (2002)