Correction: Globotriaosylceramide Accumulation and Not Alpha-Galactosidase-A Deficiency Causes Endothelial Dysfunction in Fabry Disease

Eitemau Tebyg

• Globotriaosylsphingosine Accumulation and Not Alpha-Galactosidase-A Deficiency Causes Endothelial Dysfunction in Fabry Disease
  gan: Namdar, Mehdi, et al.
  Cyhoeddwyd: (2012)
• Infusion of α-galactosidase A reduces tissue globotriaosylceramide storage in patients with Fabry disease
  gan: Schiffmann, R., et al.
  Cyhoeddwyd: (2000)
• Accumulation of Globotriaosylceramide in Podocytes in Fabry Nephropathy Is Associated with Progressive Podocyte Loss
  gan: Najafian, Behzad, et al.
  Cyhoeddwyd: (2020)
• Massive accumulation of globotriaosylceramide in various tissues from a Fabry patient with a high antibody titer against alpha-galactosidase A after 6 years of enzyme replacement therapy
  gan: Hongo, Kenichi, et al.
  Cyhoeddwyd: (2020)
• Does administration of hydroxychloroquine/amiodarone accelerate accumulation of globotriaosylceramide and globotriaosylsphingosine in Fabry mice?
  gan: Tsukimura, Takahiro, et al.
  Cyhoeddwyd: (2021)