Inhibition of Doxorubicin-Induced Senescence by PPARδ Activation Agonists in Cardiac Muscle Cells: Cooperation between PPARδ and Bcl6

Senescence and apoptosis are two distinct cellular programs that are activated in response to a variety of stresses. Low or high doses of the same stressor, i.e., the anticancer drug doxorubicin, may either induce apoptosis or senescence, respectively, in cardiac muscle cells. We have demonstrated t...

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Bibliografische gegevens
Hoofdauteurs: Altieri, Paola, Spallarossa, Paolo, Barisione, Chiara, Garibaldi, Silvano, Garuti, Anna, Fabbi, Patrizia, Ghigliotti, Giorgio, Brunelli, Claudio
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Public Library of Science 2012
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Research Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3458009/
https://ncbi.nlm.nih.gov/pubmed/23049957
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0046126
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