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Rad26p, a transcription-coupled repair factor, promotes the eviction and prevents the reassociation of histone H2A-H2B dimer during transcriptional elongation in vivo
We have recently demonstrated the formation of an atypical histone H2A–H2B dimer-enriched chromatin at the coding sequence of active gene in absence of Rad26p in vivo. However, the mechanisms for such surprising observation remain unknown. Here, using a ChIP assay, we demonstrate that Rad26p promote...
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| Hauptverfasser: | , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2012
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3447996/ https://ncbi.nlm.nih.gov/pubmed/22794311 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/bi3005768 |
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