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Enhanced Th17 differentiation and aggravated arthritis in IEX-1-deficient mice by mitochondrial ROS-mediated signaling
CD4(+) T helper (Th)1 and Th17 cells both can cause autoimmune diseases either alone or collaboratively, if left unchecked. However, what determines the dominant Th effector phenotype in a specific autoimmune disease remains ill understood. Our present investigation shows that null mutation of immed...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
2012
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3440304/ https://ncbi.nlm.nih.gov/pubmed/22798682 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1200528 |
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