Caspase-6 does not contribute to the proteolysis of mutant huntingtin in the HdhQ150 knock-in mouse model of Huntington’s disease

समान संसाधन

• Formation of Polyglutamine Inclusions in a Wide Range of Non-CNS Tissues in the HdhQ150 Knock-In Mouse Model of Huntington's Disease
  द्वारा: Moffitt, Hilary, और अन्य
  प्रकाशित: (2009)
• Correlations of Behavioral Deficits with Brain Pathology Assessed through Longitudinal MRI and Histopathology in the HdhQ150/Q150 Mouse Model of Huntington's Disease.
  द्वारा: Ivan Rattray, और अन्य
  प्रकाशित: (2017-01-01)
• Correlations of Behavioral Deficits with Brain Pathology Assessed through Longitudinal MRI and Histopathology in the HdhQ150/Q150 Mouse Model of Huntington’s Disease
  द्वारा: Rattray, Ivan, और अन्य
  प्रकाशित: (2017)
• Fragments of HdhQ150 Mutant Huntingtin Form a Soluble Oligomer Pool That Declines with Aggregate Deposition upon Aging
  द्वारा: Marcellin, David, और अन्य
  प्रकाशित: (2012)
• Mutant huntingtin gene-dose impacts on aggregate deposition, DARPP32 expression and neuroinflammation in HdhQ150 mice.
  द्वारा: Douglas Young, और अन्य
  प्रकाशित: (2013-01-01)