Neuroprotection by inhibiting the c-Jun N-terminal kinase pathway after cerebral ischemia occurs independently of interleukin-6 and keratinocyte-derived chemokine (KC/CXCL1) secretion

BACKGROUND: Cerebral ischemia is associated with the activation of glial cells, infiltration of leukocytes and an increase in inflammatory mediators in the ischemic brain and systemic circulation. How this inflammatory response influences lesion size and neurological outcome remains unclear. D-JNKI1...

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Bibliographische Detailangaben
Hauptverfasser: Benakis, Corinne, Vaslin, Anne, Pasquali, Christian, Hirt, Lorenz
Format: Artigo
Sprache:Inglês
Veröffentlicht: BioMed Central 2012
Schlagworte:
Short Report
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3416579/
https://ncbi.nlm.nih.gov/pubmed/22533966
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-9-76
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