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The flexibility of two tropomyosin mutants, D175N and E180G, that cause hypertrophic cardiomyopathy
Point mutations targeting muscle thin filament proteins are the cause of a number of cardiomyopathies. In many cases, biological effects of the mutations are well-documented, whereas their structural and mechanical impact on filament assembly and regulatory function is lacking. In order to elucidate...
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| Główni autorzy: | , , , , , , |
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| Format: | Artigo |
| Język: | Inglês |
| Wydane: |
2012
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| Hasła przedmiotowe: | |
| Dostęp online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3412897/ https://ncbi.nlm.nih.gov/pubmed/22789852 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbrc.2012.06.141 |
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