Deficits in Cognition and Synaptic Plasticity in a Mouse Model of Down Syndrome Ameliorated by GABA(B) Receptor Antagonists

समान संसाधन

• The “Down Syndrome Critical Region” Is Sufficient in the Mouse Model to Confer Behavioral, Neurophysiological, and Synaptic Phenotypes Characteristic of Down Syndrome
  द्वारा: Belichenko, Nadia P., और अन्य
  प्रकाशित: (2009)
• Comprehensive Behavioral Phenotyping of Ts65Dn Mouse Model of Down Syndrome: Activation of β(1)-Adrenergic Receptor by Xamoterol as a Potential Cognitive Enhancer
  द्वारा: Faizi, Mehrdad, और अन्य
  प्रकाशित: (2011)
• Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome
  द्वारा: Kleschevnikov, Alexander M., और अन्य
  प्रकाशित: (2017)
• Distinctions among GABA(A) and GABA(B) responses revealed by calcium channel antagonists, cannabinoids, opioids, and synaptic plasticity in rat hippocampus
  द्वारा: Lafourcade, Carlos A., और अन्य
  प्रकाशित: (2007)
• Profound deficits in hippocampal synaptic plasticity after traumatic brain injury and seizure is ameliorated by prophylactic levetiracetam
  द्वारा: Chen, Yuan-Hao, और अन्य
  प्रकाशित: (2018)